CALCIUM IN CORONARY ARTERIES
WHAT DOES IT MEAN? HOW CAN IT BE REMOVED?
(Adapted from Majid Ali's forthcoming book The
Pheasant, Oxygen, and Hypertension)
A turtle breathes and lives by what is beneath its shell.
The action is not in the shell. The action in the human
circulating blood is at the inner lining of vessel wall
called endothelium ("endo" for short). Calcium deposits in a
turtle's body belong to its shell. Calcium deposits in the
human body belong to bones.
I offer the analogy of the tire of a bicycle I used to ride
in middle school to elaborate the crucial point of the
preceding paragraph. My cycle had an inner thin-walled
rubber tube and a sturdy outer rubber tube. When the tire
went flat, I removed the inner tube, pumped air into it,
immerse it in a basin of water to detect the leak as the air
bubbled out, and then applied a rubber patch to seal. I use
this analogy to make an important point concerning vascular
calcium deposits for people with hypertension, heart
disease, and stroke: What is crucial is the functionality of
the inner endo lining ("inner tubing") of the arteries and
not calcium deposits in the outer muscle layer ("outer
tubing") of the vessel. Endo is my abbreviation of
endothelial cell that keeps the inner surface of vessels
smooth, negatively-charged, and repellent to blood cells.
So, it prevents the formation of microscopic clots on its
As long as the endo lining of the vessel retains its smooth
surface and a healthy charge, blood cells (platelets, red
blood cells, and others) do not stick to the vessel. There
is no danger of plaque buildup or the occurrence of heart
attacks or strokes. It does not matter how much calcium is
in the outer tube of the vessel. If the endo lining loses
its smooth surface and a healthy charge, blood cells will
stick to the vessel, beginning the process of plaque
formation, heart attacks, and strokes—a process that clearly
does not depend on the existence of calcium in the outer
layers of the vessel wall. To underscore this point, below
is a conversation I once had with a physician:
"Dr. Ali, I want to use scans for coronary calcium scores to
entice people for my chelation practice. What do you think?"
"It's not a good idea," I replied.
"Not good. Why?" he asked, puzzled.
"If you do that, you will have to eat crow." I smiled.
"Eat crow?" he frowned. "Don't you give chelation at your
center?" he asked with irritation.
"Yes, I do."
"Why do you do it?"
"Because it works. EDTA is extremely beneficial for patients
with coronary disease, stroke, kidney failure and other
"Then why do you say I will to have to eat crow?"
"Because it will not do what you think it will."
"Are you saying EDTA chelation does not remove calcium?"
"No, I'm not saying that."
"Then why do you say I should not promote it?"
"I'm not saying you shouldn't promote it, only that you
promote it with good arguments."
"Doesn't EDTA chelation remove calcium from coronary
"Yes, it does."
"Now you confuse me."
"With chelation you will help most of your patients within
weeks and months, but not by removing calcium from calcium
"But EDTA does remove calcium from coronary arteries,
doesn't it? Then why will I have to eat crow?"
"Because if you pull patients in with the promise of
reducing coronary calcium scores, they will want to know if
your promise was true. The main benefit of EDTA chelation in
the first year or two come from the restoration of inner
endothelium, not from removing inactive calcium deposits in
outer tissues of coronary arteries. Your patients will not
wait for three or more years to repeat their coronary
calcium scan and find out if you were right or wrong. That's
when you will have to eat crow, or maybe even a flock of
crows," I explained with a smile.
Calcium in coronary arteries is not the real issue. Calcium
deposits do not cause plaque. Rather, calcium is deposited
as a consequence of the factors that cause excess acidity,
incremental free radical activity, persistent thickening of
the circulating blood—all conditions created by lack of
oxygen. Then follows damage to endo cells, stiffening of
muscle cells in the vessels, and plaque formation.
So, the real issue is the health of the blood flowing
through arteries, not the amount of calcium in the vascular
walls. If the circulating blood is free of microclots and
micro-plaques, the vessel wall is in cleansing mode and
eventually most of the calcium in it will be reabsorbed.
Calcium left behind in very small quantities will be of no
consequence. On the other hand, if the circulating blood
carries microclots and micro-plaques, the vessel wall is in
clogging mode and additional calcium will continue to be
Dr. Ali's Guidelines for
Removal of Coronary Artery Calcium
Since calcium in coronary and other arteries is deposited
due to local tissue problems—excess acidity, oxyradical
activity, and thickening of fluids—calcium deposition can be
removed by addressing these problems. Since the root of
these problems is dysfunctional oxygen metabolism (the dysox
state), any and all measures that normalize local tissue
oxygen conditions will prevent further calcium deposits and
facilitate slow and steady removal of the deposits.
Following are four top recommendations for my patients with
calcium deposits in arteries:
Limbic Breathing and spiritual work
Castor oil rub on
abdomen and chest
breakfast (includes lecithin, flaxseed, and organic
Following are other recommendations for my patients for
calcium deposits in arteries:
"Dr. Ali's Insulin Diet Plan" (when insulin excess and
Oral chelation with DMSA, lipoic acid, and 3C combination"
(cilantro, chlorella, and chlorophyll),
Spices in rotation, such as turmeric, ginger, garlic,
onions, cumin, coriander, celery, cayenne, and others,
Herbs in rotation, including hawthorne, passionflower,
astragalus, coleus forskohlii, cat's claw, and others.
Intravenous EDTA infusions (includes supplementation with
magnesium, potassium, and taurine).
Upon beginning this program, I advise my patients a
"Begin-Low-Build-Slow Approach" in order to minimize any
initial untoward effects.
For additional details, go to
For an in-depth discussion of this subject and for details
of EDTA chelation therapies, I refer professional readers to
my book Darwin, Dysox, and Disease (2009), the
eleventh volume of The Principles and Practice of